作者
Jaclyn Carberry, David Carrick, Caroline Haig, Sam Rauhalammi, Nadeem Ahmed, Ify Mordi, Margaret B McEntegart, Mark Petrie, Hany Eteiba, Stuart Hood, Stuart Watkins, Mitchell Lindsay, Andrew Davie, Ahmed Mahrous, Ian Ford, Naveed Sattar, Paul Welsh, Keith G Oldroyd, Aleksandra Radjenovic, Colin Berry
发表日期
2016/6/1
来源
Heart
卷号
102
期号
Suppl 6
页码范围
A81-A81
出版商
BMJ Publishing Group Ltd and British Cardiovascular Society
简介
Background
The natural history of persistent myocardial oedema after ST-elevation myocardial infarction (STEMI) is uncertain.
Methods
and ResultsPatients who sustained an acute STEMI were enrolled in a cohort study (BHF MR-MI; ClinicalTrials.gov: NCT02072850). Cardiac magnetic resonance (CMR) imaging with T2-mapping of myocardial oedema was performed at 1.5 Tesla 2 days and 6 months post-MI. Myocardial oedema was defined as infarct signal intensity (S. I.) >2 standard deviations from the mean S. I. within a remote reference region. 283 STEMI patients (mean (SD) age 59 (12) years, 75% male) were enrolled. Infarct size was 18 (13)% of left ventricular (LV) mass. At 2 days, infarct zone T2 (ms) was higher than remote zone T2 (62.9 (5.2) vs. 49.7 (2.1); p < 0.001), and this relationship persisted at 6 months (56.2 (4.1) vs. 49.7 (2.3); p < 0.001). Mean remote zone T2 did not change over time (p = 0.840 …
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