| The process of Lewy body formation, rather than simply α-synuclein fibrillization, is one of the major drivers of neurodegeneration AL Mahul-Mellier, J Burtscher, N Maharjan, L Weerens, M Croisier, ... Proceedings of the National Academy of Sciences 117 (9), 4971-4982, 2020 | 572 | 2020 |
| C9ORF72 regulates stress granule formation and its deficiency impairs stress granule assembly, hypersensitizing cells to stress N Maharjan, C Künzli, K Buthey, S Saxena Molecular neurobiology 54, 3062-3077, 2017 | 118 | 2017 |
| Marinesco-Sjögren syndrome protein SIL1 regulates motor neuron subtype-selective ER stress in ALS A Filézac de L'Etang, N Maharjan, M Cordeiro Braña, C Ruegsegger, ... Nature neuroscience 18 (2), 227-238, 2015 | 108 | 2015 |
| Nuclear and cytoplasmic huntingtin inclusions exhibit distinct biochemical composition, interactome and ultrastructural properties N Riguet, AL Mahul-Mellier, N Maharjan, J Burtscher, M Croisier, G Knott, ... Nature communications 12 (1), 6579, 2021 | 62 | 2021 |
| Aberrant association of misfolded SOD1 with Na+/K+ATPase-α3 impairs its activity and contributes to motor neuron vulnerability in ALS C Ruegsegger, N Maharjan, A Goswami, A Filézac de L’Etang, J Weis, ... Acta neuropathologica 131, 427-451, 2016 | 56 | 2016 |
| Emerging perspectives on dipeptide repeat proteins in C9ORF72 ALS/FTD A Schmitz, J Pinheiro Marques, I Oertig, N Maharjan, S Saxena Frontiers in cellular neuroscience 15, 637548, 2021 | 46 | 2021 |
| The making of a Lewy body: the role of α-synuclein post-fibrillization modifications in regulating the formation and the maturation of pathological inclusions AL Mahul-Mellier, MF Altay, J Burtscher, N Maharjan, N Ait-Bouziad, ... BioRxiv, 500058, 2018 | 45 | 2018 |
| P2X1‐regulated IL‐22 secretion by innate lymphoid cells is required for efficient liver regeneration R Kudira, T Malinka, A Kohler, M Dosch, MG de Agüero, N Melin, ... Hepatology 63 (6), 2004-2017, 2016 | 41 | 2016 |
| Seeding the aggregation of TDP-43 requires post-fibrillization proteolytic cleavage ST Kumar, S Nazarov, S Porta, N Maharjan, U Cendrowska, M Kabani, ... Nature Neuroscience 26 (6), 983-996, 2023 | 24 | 2023 |
| Filezac de L’Etang, A., Weis, J., Troost, D., et al.(2016). Aberrant association of misfolded SOD1 with Na (+)/K (+) ATPase-alpha3 impairs its activity and contributes to motor … C Ruegsegger, N Maharjan, A Goswami Acta Neuropathol 131, 427-451, 0 | 18 | |
| ER strikes again: proteostasis dysfunction in ALS N Maharjan, S Saxena The EMBO journal 35 (8), 798-800, 2016 | 14 | 2016 |
| PolyGA targets the ER stress-adaptive response by impairing GRP75 function at the MAM in C9ORF72-ALS/FTD F Pilotto, A Schmitz, N Maharjan, R Diab, A Odriozola, P Tripathi, ... Acta neuropathologica 144 (5), 939-966, 2022 | 12 | 2022 |
| Disentangling the sequence, cellular and ultrastructural determinants of Huntingtin nuclear and cytoplasmic inclusion formation N Riguet, AL Mahul-Mellier, N Maharjan, J Burtscher, A Patin, M Croisier, ... bioRxiv, 2020 | 11 | 2020 |
| Sphingolipids and impaired hypoxic stress responses in Huntington disease J Burtscher, G Pepe, N Maharjan, N Riguet, A Di Pardo, V Maglione, ... Progress in lipid research 90, 101224, 2023 | 8 | 2023 |
| It takes two to tango: DPRs in ALS and SCA36 N Maharjan, S Saxena Neuron 107 (2), 202-204, 2020 | 4 | 2020 |
| The process of Lewy body formation, rather than simply alpha-synuclein fibrillization, is the major driver of neurodegeneration in synucleinopathies AL Mahul-Mellier, J Burtscher, N Maharjan, L Weerens, M Croisier, ... BiorXiv, 751891, 2019 | 4 | 2019 |
| Models of Neurodegenerative Diseases N Maharjan, S Saxena Neurogenetics: Current Topics in Cellular and Developmental Neurobiology …, 2022 | | 2022 |
| Investigating the Function of C9ORF72; Protein Product of a Novel Gene Implicated in Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal Lobar Degeneration (FTLD) N Maharjan Medizinische Fakultät der Universität Bern, 2015 | | 2015 |
| LMNN MF Altay, A Ansaloni, L Awad, N Aït Bouziad, MT Baillie, RI Benmessaoud, ... | | |
| The dynamics of Lewy body formation, rather than simply alpha-synuclein fibrillization, is the primary cause of mitochondrial alterations, synaptic dysfunction and … AL Mahul-Mellier, J Burtscher, N Maharjan, L Weerens, M Croisier, ... | | |
