受强制性开放获取政策约束的文章 - marjorie maillet了解详情
可在其他位置公开访问的文章：19 篇
Molecular basis of physiological heart growth: fundamental concepts and new players
M Maillet, JH Van Berlo, JD Molkentin
Nature reviews Molecular cell biology 14 (1), 38-48, 2013
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
C-kit+ cells minimally contribute cardiomyocytes to the heart
JH Van Berlo, O Kanisicak, M Maillet, RJ Vagnozzi, J Karch, SCJ Lin, ...
Nature 509 (7500), 337-341, 2014
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
Signaling effectors underlying pathologic growth and remodeling of the heart
JH van Berlo, M Maillet, JD Molkentin
The Journal of clinical investigation 123 (1), 37, 2013
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
Extracellular Signal-Regulated Kinases 1 and 2 Regulate the Balance Between Eccentric and Concentric Cardiac GrowthNovelty and Significance
I Kehat, J Davis, M Tiburcy, F Accornero, MK Saba-El-Leil, M Maillet, ...
Circulation research 108 (2), 176-183, 2011
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute, German …
Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism
DP Millay, SA Goonasekera, MA Sargent, M Maillet, BJ Aronow, ...
Proceedings of the National Academy of Sciences 106 (45), 19023-19028, 2009
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
A thrombospondin-dependent pathway for a protective ER stress response
JM Lynch, M Maillet, D Vanhoutte, A Schloemer, MA Sargent, NS Blair, ...
Cell 149 (6), 1257-1268, 2012
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
DUSP6 (MKP3) null mice show enhanced ERK1/2 phosphorylation at baseline and increased myocyte proliferation in the heart affecting disease susceptibility
M Maillet, NH Purcell, MA Sargent, AJ York, OF Bueno, JD Molkentin
Journal of Biological Chemistry 283 (45), 31246-31255, 2008
强制性开放获取政策： US National Institutes of Health
The IP3 Receptor Regulates Cardiac Hypertrophy in Response to Select StimuliNovelty and Significance
H Nakayama, I Bodi, M Maillet, J DeSantiago, TL Domeier, K Mikoshiba, ...
Circulation research 107 (5), 659-666, 2010
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
A Critical Function for Ser-282 in Cardiac Myosin Binding Protein-C Phosphorylation and Cardiac FunctionNovelty and Significance
S Sadayappan, J Gulick, H Osinska, D Barefield, F Cuello, M Avkiran, ...
Circulation research 109 (2), 141-150, 2011
强制性开放获取政策： US National Institutes of Health
Lost in Transgenesis
J Davis, M Maillet, JM Miano, JD Molkentin
Circulation research 111 (6), 761-777, 2012
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
Six1 regulates stem cell repair potential and self-renewal during skeletal muscle regeneration
F Le Grand, R Grifone, P Mourikis, C Houbron, C Gigaud, J Pujol, ...
J Cell Biol 198 (5), 815-832, 2012
强制性开放获取政策： National Institute of Health and Medical Research, France
Tinman/Nkx2-5 acts via miR-1 and upstream of Cdc42 to regulate heart function across species
L Qian, JD Wythe, J Liu, J Cartry, G Vogler, B Mohapatra, RT Otway, ...
The Journal of cell biology, jcb. 201006114, 2011
强制性开放获取政策： US National Institutes of Health
Mutual antagonism between IP3RII and miRNA-133a regulates calcium signals and cardiac hypertrophy
FM Drawnel, D Wachten, JD Molkentin, M Maillet, JM Aronsen, F Swift, ...
The Journal of General Physiology 141 (1), i1-i1, 2013
强制性开放获取政策： British Heart Foundation
Cdc42 is an antihypertrophic molecular switch in the mouse heart
M Maillet, JM Lynch, B Sanna, AJ York, Y Zheng, JD Molkentin
The Journal of clinical investigation 119 (10), 3079, 2009
强制性开放获取政策： US National Institutes of Health
DUSP8 Regulates Cardiac Ventricular Remodeling by Altering ERK1/2 SignalingNovelty and Significance
R Liu, JH van Berlo, AJ York, RJ Vagnozzi, M Maillet, JD Molkentin
Circulation research 119 (2), 249-260, 2016
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
Heart-specific deletion of CnB1 reveals multiple mechanisms whereby calcineurin regulates cardiac growth and function
M Maillet, J Davis, M Auger-Messier, A York, H Osinska, J Piquereau, ...
Journal of Biological Chemistry 285 (9), 6716-6724, 2010
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
Genetic manipulation of dysferlin expression in skeletal muscle: novel insights into muscular dystrophy
DP Millay, M Maillet, JA Roche, MA Sargent, EM McNally, RJ Bloch, ...
The American journal of pathology 175 (5), 1817-1823, 2009
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute
Cathepsin S contributes to the pathogenesis of muscular dystrophy in mice
A Tjondrokoesoemo, TG Schips, MA Sargent, D Vanhoutte, O Kanisicak, ...
Journal of Biological Chemistry 291 (19), 9920-9928, 2016
强制性开放获取政策： US National Institutes of Health, Howard Hughes Medical Institute, American …
Spatial Gene Profiling in the Ischemic Heart: Fibroblasts Put on Their SOX
H Khalil, M Maillet, JD Molkentin
Circulation 136 (15), 1410-1411, 2017
强制性开放获取政策： Swiss National Science Foundation, US National Institutes of Health, Howard …
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