受强制性开放获取政策约束的文章 - Ognian Ikonomov了解详情
可在其他位置公开访问的文章：22 篇
The phosphoinositide kinase PIKfyve is vital in early embryonic development: preimplantation lethality of PIKfyve−/− embryos but normality of PIKfyve+/− mice
OC Ikonomov, D Sbrissa, K Delvecchio, Y Xie, JP Jin, D Rappolee, ...
Journal of Biological Chemistry 286 (15), 13404-13413, 2011
强制性开放获取政策： US National Institutes of Health
Molecular mechanisms underlying mood stabilization in manic-depressive illness: the phenotype challenge
OC Ikonomov, HK Manji
Bipolar Disorder, 194-202, 2019
强制性开放获取政策： US National Institutes of Health
ArPIKfyve homomeric and heteromeric interactions scaffold PIKfyve and Sac3 in a complex to promote PIKfyve activity and functionality
D Sbrissa, OC Ikonomov, H Fenner, A Shisheva
Journal of molecular biology 384 (4), 766-779, 2008
强制性开放获取政策： US National Institutes of Health
Functional dissociation between PIKfyve-synthesized PtdIns5P and PtdIns(3,5)P2 by means of the PIKfyve inhibitor YM201636
D Sbrissa, OC Ikonomov, C Filios, K Delvecchio, A Shisheva
American Journal of Physiology-Cell Physiology 303 (4), C436-C446, 2012
强制性开放获取政策： US National Institutes of Health
PIKfyve-ArPIKfyve-Sac3 Core Complex
OC Ikonomov, D Sbrissa, H Fenner, A Shisheva
Journal of Biological Chemistry 284 (51), 35794-35806, 2009
强制性开放获取政策： US National Institutes of Health
YM201636, an inhibitor of retroviral budding and PIKfyve-catalyzed PtdIns (3, 5) P2 synthesis, halts glucose entry by insulin in adipocytes
OC Ikonomov, D Sbrissa, A Shisheva
Biochemical and biophysical research communications 382 (3), 566-570, 2009
强制性开放获取政策： US National Institutes of Health
ArPIKfyve regulates Sac3 protein abundance and turnover: disruption of the mechanism by Sac3I41T mutation causing Charcot-Marie-Tooth 4J disorder
OC Ikonomov, D Sbrissa, J Fligger, K Delvecchio, A Shisheva
Journal of Biological Chemistry 285 (35), 26760-26764, 2010
强制性开放获取政策： US National Institutes of Health
Small molecule PIKfyve inhibitors as cancer therapeutics: Translational promises and limitations
OC Ikonomov, D Sbrissa, A Shisheva
Toxicology and Applied Pharmacology 383, 114771, 2019
强制性开放获取政策： US Department of Defense, US National Institutes of Health
The PIKfyve–ArPIKfyve–Sac3 triad in human breast cancer: Functional link between elevated Sac3 phosphatase and enhanced proliferation of triple negative cell lines
OC Ikonomov, C Filios, D Sbrissa, X Chen, A Shisheva
Biochemical and biophysical research communications 440 (2), 342-347, 2013
强制性开放获取政策： US National Institutes of Health
Active vacuolar H+ ATPase and functional cycle of Rab5 are required for the vacuolation defect triggered by PtdIns(3,5)P2 loss under PIKfyve or Vps34 deficiency
LM Compton, OC Ikonomov, D Sbrissa, P Garg, A Shisheva
American Journal of Physiology-Cell Physiology 311 (3), C366-C377, 2016
强制性开放获取政策： US National Institutes of Health
Kinesin adapter JLP links PIKfyve to microtubule-based endosome-to-trans-Golgi network traffic of furin
OC Ikonomov, J Fligger, D Sbrissa, R Dondapati, K Mlak, R Deeb, ...
Journal of Biological Chemistry 284 (6), 3750-3761, 2009
强制性开放获取政策： US National Institutes of Health
Sac3 is an insulin-regulated phosphatidylinositol 3, 5-bisphosphate phosphatase: gain in insulin responsiveness through Sac3 down-regulation in adipocytes
OC Ikonomov, D Sbrissa, T Ijuin, T Takenawa, A Shisheva
Journal of Biological Chemistry 284 (36), 23961-23971, 2009
强制性开放获取政策： US National Institutes of Health
Apilimod, a candidate anticancer therapeutic, arrests not only PtdIns(3,5)P2 but also PtdIns5P synthesis by PIKfyve and induces bafilomycin A1-reversible aberrant …
D Sbrissa, G Naisan, OC Ikonomov, A Shisheva
PloS one 13 (9), e0204532, 2018
强制性开放获取政策： US Department of Defense, US National Institutes of Health
Class III PI 3-kinase is the main source of PtdIns3P substrate and membrane recruitment signal for PIKfyve constitutive function in podocyte endomembrane homeostasis
OC Ikonomov, D Sbrissa, M Venkatareddy, E Tisdale, P Garg, A Shisheva
Biochimica et Biophysica Acta (BBA)-Molecular Cell Research 1853 (5), 1240-1250, 2015
强制性开放获取政策： US National Institutes of Health
Plentiful PtdIns5P from scanty PtdIns(3,5)P2 or from ample PtdIns? PIKfyve‐dependent models: Evidence and speculation (response to: DOI 10.1002/bies …
A Shisheva, D Sbrissa, O Ikonomov
Bioessays 37 (3), 267-277, 2015
强制性开放获取政策： US National Institutes of Health, Juvenile Diabetes Research Foundation
Muscle-specific Pikfyve gene disruption causes glucose intolerance, insulin resistance, adiposity, and hyperinsulinemia but not muscle fiber-type switching
OC Ikonomov, D Sbrissa, K Delvecchio, HZ Feng, GD Cartee, JP Jin, ...
American Journal of Physiology-Endocrinology and Metabolism 305 (1), E119-E131, 2013
强制性开放获取政策： US National Institutes of Health
Host PI(3,5)P2 Activity Is Required for Plasmodium berghei Growth During Liver Stage Infection
C Thieleke‐Matos, ML da Silva, L Cabrita‐Santos, CF Pires, JS Ramalho, ...
Traffic 15 (10), 1066-1082, 2014
强制性开放获取政策： US National Institutes of Health, Fundação para a Ciência e a Tecnologia …
PIKfyve inhibitor cytotoxicity requires AKT suppression and excessive cytoplasmic vacuolation
OC Ikonomov, G Altankov, D Sbrissa, A Shisheva
Toxicology and applied pharmacology 356, 151-158, 2018
强制性开放获取政策： US Department of Defense
Unexpected severe consequences of Pikfyve deletion by aP2‐ or Aq‐promoter‐driven Cre expression for glucose homeostasis and mammary gland development
OC Ikonomov, D Sbrissa, K Delvecchio, J A. Rillema, A Shisheva
Physiological Reports 4 (11), e12812, 2016
强制性开放获取政策： US National Institutes of Health
The protein complex of neurodegeneration-related phosphoinositide phosphatase Sac3 and ArPIKfyve binds the Lewy body-associated Synphilin-1, preventing its aggregation
OC Ikonomov, D Sbrissa, LM Compton, R Kumar, EJ Tisdale, X Chen, ...
Journal of Biological Chemistry 290 (47), 28515-28529, 2015
强制性开放获取政策： US National Institutes of Health
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