Alcohol consumption, in particular ethanol (EtOH), typically begins in human adolescence,
often in a “binge like” manner. However, although EtOH abuse has a high prevalence at this …

Ethanol (EtOH) intake and noise exposure are particularly concerning among human
adolescents because the potential to harm brain. Unfortunately, putative underlying …

Background The consumption of alcohol during pregnancy can result in abnormal fetal
development and impaired brain function in humans and experimental animal models …

Background: The cerebellum of the neonatal rat is highly susceptible to ethanol, with
profound loss of Purkinje cells resulting from even brief exposure during the first postnatal …

Ethanol exposure during pregnancy can produce a variety of central nervous system
abnormalities in the offspring, resulting in a broad spectrum of cognitive and behavioral …

Background Ethanol (EtOH), one of the most widely consumed substances of abuse, can
induce brain damage and neurodegeneration. Et OH is centrally metabolized into …

Ethanol‐induced neuro‐developmental abnormalities are associated with impaired insulin
and IGF signaling, and increased oxidative stress in CNS neurons. We examined the roles …

Oxidative stress is a candidate mechanism for ethanol neuropathology in fetal alcohol
spectrum disorders. Oxidative stress often involves production of reactive oxygen species …

Alcohol intake by women before planning a pregnancy, during pregnancy, and/or during
lactation induces brain, behavioral, and physical abnormalities in the fetus, which are known …

Free radicals species generation during ethanol metabolism is implicated in ethanol-
induced toxicity. Findings from clinical studies have clearly established the association …