Abstract Coronavirus disease 2019 (COVID-19) is a potentially life-threatening infection
characterized by excessive inflammation, coagulation disorders and organ damage. A
dysregulated myeloid cell compartment is one of the most striking immunopathologic
signatures of this newly emerged infection. A growing number of studies are reporting on the
expansion of myeloid cells with immunoregulatory activities in the periphery and airways of
COVID-19 patients. These cells share phenotypic and functional similarities with myeloid …