[HTML][HTML] TAF1 bromodomain inhibition as a candidate epigenetic driver of congenital heart disease
RS Leigh, MJ Välimäki, BL Kaynak… - Biochimica et Biophysica …, 2023 - Elsevier
Heart formation requires transcriptional regulators that underlie congenital anomalies and
the fetal gene program activated during heart failure. Attributing the effects of congenital …
the fetal gene program activated during heart failure. Attributing the effects of congenital …
[PDF][PDF] BBA-Molecular Basis of Disease
RS Leigh, MJ Välimäki, BL Kaynak, HJ Ruskoaho - tuhat.helsinki.fi
Heart formation requires transcriptional regulators that underlie congenital anomalies and
the fetal gene program activated during heart failure. Attributing the effects of congenital …
the fetal gene program activated during heart failure. Attributing the effects of congenital …
[PDF][PDF] TAF1 bromodomain inhibition as a candidate epigenetic driver of congenital heart disease
R Leigh - helda.helsinki.fi
Heart formation requires transcriptional regulators that underlie congenital anomalies and
the fetal gene program activated during heart failure. Attributing the effects of congenital …
the fetal gene program activated during heart failure. Attributing the effects of congenital …
[PDF][PDF] TAF1 bromodomain inhibition as a candidate epigenetic driver of congenital heart disease
R Leigh, M Välimäki, B Kaynak… - … et Biophysica Acta …, 2023 - researchportal.helsinki.fi
Heart formation requires transcriptional regulators that underlie congenital anomalies and
the fetal gene program activated during heart failure. Attributing the effects of congenital …
the fetal gene program activated during heart failure. Attributing the effects of congenital …
TAF1 bromodomain inhibition as a candidate epigenetic driver of congenital heart disease
RS Leigh, MJ Välimäki, BL Kaynak… - … et biophysica acta …, 2023 - pubmed.ncbi.nlm.nih.gov
Heart formation requires transcriptional regulators that underlie congenital anomalies and
the fetal gene program activated during heart failure. Attributing the effects of congenital …
the fetal gene program activated during heart failure. Attributing the effects of congenital …
TAF1 bromodomain inhibition as a candidate epigenetic driver of congenital heart disease.
RS Leigh, MJ Välimäki, BL Kaynak… - … et Biophysica acta …, 2023 - europepmc.org
Heart formation requires transcriptional regulators that underlie congenital anomalies and
the fetal gene program activated during heart failure. Attributing the effects of congenital …
the fetal gene program activated during heart failure. Attributing the effects of congenital …
[引用][C] TAF1 bromodomain inhibition as a candidate epigenetic driver of congenital heart disease
R Leigh, M Välimäki, B Kaynak, H Ruskoaho - 2023 - helda.helsinki.fi