The core pathology of coronavirus disease 2019 (COVID-19) is infection of airway cells by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) that results in excessive …
S Turner, CA Naidoo, TJ Usher… - … in Thrombosis and …, 2024 - thieme-connect.com
The prevailing hypotheses for the persistent symptoms of Long COVID have been narrowed down to immune dysregulation and autoantibodies, widespread organ damage, viral …
JD McFadyen, H Stevens, K Peter - Circulation research, 2020 - Am Heart Assoc
The recent emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and the ensuing global pandemic has presented a health emergency of unprecedented …
H Shi, Y Zuo, S Navaz, A Harbaugh… - Arthritis & …, 2022 - Wiley Online Library
Objective While endothelial dysfunction has been implicated in the widespread thromboinflammatory complications of COVID‐19, the upstream mediators of …
Abstract Coronavirus disease 2019 (COVID-19) is a clinical syndrome caused by infection with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Patients with severe …
CMAJ| OCTOBER 5, 2020| VOLUME 192| ISSUE 40 E1157 in thrombotic microangiopathy by inducing tissue factor expression, secretion of von Willebrand factor, and secretion and …
Introduction Endothelial damage and thrombosis caused by COVID-19 may imperil cardiovascular health. More than a year since the WHO declared COVID-19 pandemic …
MJ Al-Farabi, RA Nugraha, BA Marsudi, Y Azmi - Microvascular research, 2021 - Elsevier
Background Several studies have reported that the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) can directly infect endothelial cells, and endothelial …
Patients with coronavirus disease 2019 (COVID-19) who are critically ill develop vascular complications characterized by thrombosis of small, medium, and large vessels. Dysfunction …