Activation of extracellular signal-regulated kinase1/2 in the medial prefrontal cortex contributes to stress-induced hyperalgesia

J Qi, C Chen, YC Lu, T Zhang, H Xu, YY Cui… - Molecular …, 2014 - Springer
Stressful stimuli can exacerbate persistent pain disorder. However, the underlying
mechanism is still unknown. Here, to reveal the underlying mechanism for stressful stimuli …
被引用次数：20 相关文章

Activation of extracellular signal-regulated kinase1/2 in the medial prefrontal cortex contributes to stress-induced hyperalgesia

J Qi, C Chen, YC Lu, T Zhang, H Xu… - Molecular …, 2014 - pubmed.ncbi.nlm.nih.gov
Stressful stimuli can exacerbate persistent pain disorder. However, the underlying
mechanism is still unknown. Here, to reveal the underlying mechanism for stressful stimuli …

Activation of Extracellular Signal-Regulated Kinase1/2 in the Medial Prefrontal Cortex Contributes to Stress-Induced Hyperalgesia

J Qi, C Chen, Y Lu, T Zhang, H Xu, Y Cui… - Molecular …, 2014 - search.proquest.com
Stressful stimuli can exacerbate persistent pain disorder. However, the underlying
mechanism is still unknown. Here, to reveal the underlying mechanism for stressful stimuli …

Activation of extracellular signal-regulated kinase1/2 in the medial prefrontal cortex contributes to stress-induced hyperalgesia.

J Qi, C Chen, YC Lu, T Zhang, H Xu, YY Cui… - Molecular …, 2014 - europepmc.org
Stressful stimuli can exacerbate persistent pain disorder. However, the underlying
mechanism is still unknown. Here, to reveal the underlying mechanism for stressful stimuli …

Activation of Extracellular Signal-Regulated Kinase1/2 in the Medial Prefrontal Cortex Contributes to Stress-Induced Hyperalgesia

J Qi, C Chen, YC Lu, T Zhang, H Xu, YY Cui… - Molecular …, 2014 - infona.pl
Stressful stimuli can exacerbate persistent pain disorder. However, the underlying
mechanism is still unknown. Here, to reveal the underlying mechanism for stressful stimuli …

[引用][C] Activation of Extracellular Signal-Regulated Kinase1

J Qi, C Chen, YC Lu, T Zhang, H Xu, YY Cui… - Molecular …, 2014 - Springer