Blockade of receptor‐activated Gi signaling in osteoblasts in vivo leads to site‐specific increases in cortical and cancellous bone formation

SM Millard, AM Louie, L Wattanachanya… - Journal of bone and …, 2011 - academic.oup.com
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered Gi …

[PDF][PDF] Blockade of Receptor-Activated Gi Signaling in Osteoblasts In Vivo Leads to Site-Specific Increases in Cortical and Cancellous Bone Formation

SM Millard, AM Louie, L Wattanachanya… - Journal of Bone and …, 2011 - researchgate.net
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered Gi …

Blockade of receptor‐activated Gi signaling in osteoblasts in vivo leads to site‐specific increases in cortical and cancellous bone formation

SM Millard, AM Louie, L Wattanachanya… - Journal of Bone and …, 2011 - escholarship.org
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered G (i) …

Blockade of receptor-activated G (i) signaling in osteoblasts in vivo leads to site-specific increases in cortical and cancellous bone formation

SM Millard, AM Louie… - Journal of bone …, 2011 - pubmed.ncbi.nlm.nih.gov
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered G (i) …

[引用][C] Blockade of Receptor-Activated Gi Signaling in Osteoblasts In Vivo Leads to Site-Specific Increases in Cortical and Cancellous Bone Formation

SM MILLARD, AM LOUIE… - Journal of bone and …, 2011 - pascal-francis.inist.fr
Blockade of Receptor-Activated Gi Signaling in Osteoblasts In Vivo Leads to Site-Specific
Increases in Cortical and Cancellous Bone Formation CNRS Inist Pascal-Francis CNRS …

Blockade of receptor-activated G (i) signaling in osteoblasts in vivo leads to site-specific increases in cortical and cancellous bone formation.

SM Millard, AM Louie, L Wattanachanya… - Journal of Bone and …, 2011 - europepmc.org
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered G i …

[HTML][HTML] Blockade of Receptor-Activated Gi Signaling in Osteoblasts In Vivo Leads to Site-Specific Increases in Cortical and Cancellous Bone Formation

SM Millard, AM Louie, L Wattanachanya… - Journal of Bone and …, 2011 - ncbi.nlm.nih.gov
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered G i …

[PDF][PDF] Blockade of Receptor-Activated Gi Signaling in Osteoblasts In Vivo Leads to Site-Specific Increases in Cortical and Cancellous Bone Formation

SM Millard, AM Louie, L Wattanachanya… - Journal of Bone and …, 2011 - Citeseer
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered Gi …

[引用][C] Blockade of receptor-activated G (i) signaling in osteoblasts in vivo leads to site-specific increases in cortical and cancellous bone formation

S Millard, AM Louie… - Journal of Bone …, 2011 - espace.library.uq.edu.au
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered Gi …

Blockade of receptor‐activated Gi signaling in osteoblasts in vivo leads to site‐specific increases in cortical and cancellous bone formation

SM Millard, AM Louie, L Wattanachanya… - Journal of Bone and …, 2011 - Wiley Online Library
Osteoblasts play a critical role in the maintenance of bone mass through bone formation and
regulation of bone resorption. Targeted expression of a constitutively active engineered Gi …