Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro
TK Kerppola, D Luk, T Curran - Molecular and Cellular Biology, 1993 - Taylor & Francis
Several regulatory interactions between the AP-1 and the nuclear hormone receptor families
of transcription factors have been reported. However, the molecular mechanisms that …
of transcription factors have been reported. However, the molecular mechanisms that …
Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro.
TK Kerppola, D Luk, T Curran - Molecular and Cellular Biology, 1993 - ncbi.nlm.nih.gov
Several regulatory interactions between the AP-1 and the nuclear hormone receptor families
of transcription factors have been reported. However, the molecular mechanisms that …
of transcription factors have been reported. However, the molecular mechanisms that …
[PDF][PDF] Fos Is Preferential Target of Glucocorticoid Receptor Inhibition of AP-1 Activity In Vitro
TOMK KERPPOLA, D LUK, TOM CURRAN - … AND CELLULAR BIOLOGY, 1993 - Citeseer
MATERIALS AND METHODS Protein expression and purification. Synthetic genes encod-
ing GR residues 440 to 533, 440 to 525, 440 to 517, and 440 to 509 were constructed by …
ing GR residues 440 to 533, 440 to 525, 440 to 517, and 440 to 509 were constructed by …
Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro.
TK Kerppola, D Luk, T Curran - Molecular and Cellular Biology, 1993 - cir.nii.ac.jp
抄録< jats: p> Several regulatory interactions between the AP-1 and the nuclear hormone
receptor families of transcription factors have been reported. However, the molecular …
receptor families of transcription factors have been reported. However, the molecular …
Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro
TK Kerppola, D Luk, T Curran - Molecular and cellular …, 1993 - pubmed.ncbi.nlm.nih.gov
Several regulatory interactions between the AP-1 and the nuclear hormone receptor families
of transcription factors have been reported. However, the molecular mechanisms that …
of transcription factors have been reported. However, the molecular mechanisms that …
[PDF][PDF] Fos Is Preferential Target of Glucocorticoid Receptor Inhibition of AP-1 Activity In Vitro
TOMK KERPPOLA, D LUK… - MOLECULAR AND …, 1993 - researchgate.net
MATERIALS AND METHODS Protein expression and purification. Synthetic genes encod-
ing GR residues 440 to 533, 440 to 525, 440 to 517, and 440 to 509 were constructed by …
ing GR residues 440 to 533, 440 to 525, 440 to 517, and 440 to 509 were constructed by …
Fos is a Preferential Target of Glucocorticoid Receptor Inhibition of AP-1 Activity In Vitro
TK Kerppola, D Luk, T Curran - Molecular and Cellular Biology, 1993 - Am Soc Microbiol
Several regulatory interactions between the AP-1 and the nuclear hormone receptor families
of transcription factors have been reported. However, the molecular mechanisms that …
of transcription factors have been reported. However, the molecular mechanisms that …
[引用][C] Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro
TK KERPPOLA, D LUK… - Molecular and cellular …, 1993 - pascal-francis.inist.fr
Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro CNRS
Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases Simple search …
Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases Simple search …
Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro.
TK Kerppola, D Luk, T Curran - Molecular and Cellular Biology, 1993 - europepmc.org
Several regulatory interactions between the AP-1 and the nuclear hormone receptor families
of transcription factors have been reported. However, the molecular mechanisms that …
of transcription factors have been reported. However, the molecular mechanisms that …
[引用][C] Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro
TK KERPPOLA - Mol Cell Biol, 1993 - cir.nii.ac.jp