Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R Terayama, S Omura, N Fujisawa, T Yamaai… - Neuroscience, 2008 - Elsevier
The activation of glial cells in the CNS has been suggested to be involved in abnormal pain
sensation after peripheral nerve injury. Previous studies demonstrated phosphorylation of …

[引用][C] ACTIVATION OF MICROGLIA AND P38 MITOGEN-ACTIVATED PROTEIN KINASE IN THE DORSAL COLUMN NUCLEUS CONTRIBUTES TO TACTILE …

R TERAYAMA, S OMURA, N FUJISAWA… - …, 2008 - pascal-francis.inist.fr
ACTIVATION OF MICROGLIA AND P38 MITOGEN-ACTIVATED PROTEIN KINASE IN THE
DORSAL COLUMN NUCLEUS CONTRIBUTES TO TACTILE ALLODYNIA FOLLOWING …

Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R Terayama, S Omura, N Fujisawa… - …, 2008 - pubmed.ncbi.nlm.nih.gov
The activation of glial cells in the CNS has been suggested to be involved in abnormal pain
sensation after peripheral nerve injury. Previous studies demonstrated phosphorylation of …

Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R Terayama, S Omura, N Fujisawa, T Yamaai… - Neuroscience, 2008 - infona.pl
The activation of glial cells in the CNS has been suggested to be involved in abnormal pain
sensation after peripheral nerve injury. Previous studies demonstrated phosphorylation of …

Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury.

R Terayama, S Omura, N Fujisawa, T Yamaai… - …, 2008 - europepmc.org
The activation of glial cells in the CNS has been suggested to be involved in abnormal pain
sensation after peripheral nerve injury. Previous studies demonstrated phosphorylation of …

Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R Terayama, S Omura, N Fujisawa… - …, 2008 - okayama.elsevierpure.com
抄録 The activation of glial cells in the CNS has been suggested to be involved in abnormal
pain sensation after peripheral nerve injury. Previous studies demonstrated phosphorylation …

[引用][C] Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R TERAYAMA - Neuroscience, 2008 - cir.nii.ac.jp
Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus
contributes to tactile allodynia following peripheral nerve injury | CiNii Research CiNii 国立 …

Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R Terayama, S Omura, N Fujisawa, T Yamaai… - …, 2008 - ibroneuroscience.org
The activation of glial cells in the CNS has been suggested to be involved in abnormal pain
sensation after peripheral nerve injury. Previous studies demonstrated phosphorylation of …

Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R Terayama, S Omura, N Fujisawa… - …, 2008 - okayama.elsevierpure.com
The activation of glial cells in the CNS has been suggested to be involved in abnormal pain
sensation after peripheral nerve injury. Previous studies demonstrated phosphorylation of …

[引用][C] Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column nucleus contributes to tactile allodynia following peripheral nerve injury

R Terayama, S Omura, N Fujisawa, T Yamaai… - Neuroscience, 2008 - cir.nii.ac.jp
Activation of microglia and p38 mitogen-activated protein kinase in the dorsal column
nucleus contributes to tactile allodynia following peripheral nerve injury | CiNii Research …