previously reported as highly resistant to dinitroaniline herbicides mainly due to a Val‐202‐
Phe substitution in the target site α‐tubulin protein. To further determine the contribution of
the 202 mutation to resistance, two sub‐populations, respectively comprising the 202 mutant
and wild‐type (WT) individuals, were isolated from within the same resistant population and
subject to dinitroaniline herbicide doses. A rice transgenic study was conducted to …