Toxoplasmosis caused by an obligatory intracellular protozoan parasite of Toxoplasma
gondii threats a wide spectrum of human and animal hosts. It has been shown that the
intensity of the disease in humans depends on the host's immune responses. Immunological
investigations on whole protein molecules of T. gondii have shown that these antigens are
not fully responsible for the immune response, which leads to a decrease in specificity and
affinity of the antigen (epitope)-antibody (paratope) binding. Currently, epitopes have shown …