Objectives: Hypertension exerts a chronic tensile stretch on the endothelial lining of the
inner layer of blood vessels and is thought to cause cellular injury leading to numerous
pathologies. Studies have indicated that tensile stretch could alter the phenotype and
cellular functions of the cell, depending on the stretch magnitude applied. However, the
molecular mechanisms of the cellular injury under these conditions remain unclear. Method:
In order to investigate the protein changes potentially involved in hypertension, human …