mainly causes alterations in the respiratory system. In severely ill patients, the disease often
evolves into an acute respiratory distress syndrome that can predispose patients to a state of
hypercoagulability, with thrombosis at both venous and arterial levels. This predisposition
presents a multifactorial physiopathology, related to hypoxia as well as to the severe
inflammatory process linked to this pathology, including the additional thrombotic factors …