Spontaneous coronary artery dissection (SCAD) remains an infrequent, elusive, and
challenging clinical entity of unknown etiology eight decades after its initial description. Our
understanding of the pathophysiology of SCAD, initially limited to information from early
pathological studies, case reports, and very short series, has been enriched recently by
relatively large contemporary series of patients studied prospectively. The typical
presentation involves a young woman without coronary risk factors suffering an acute …