… brain injury after TBI is essential for the identification of new potential therapeutic targets.
Peroxisome proliferator … In 1987, a peroxisome proliferator-binding protein was identified and …

… While the primary injury results from mechanical forces applied to the skull and brain at the
time of impact, secondary brain injury … Secondary brain injuries are mediated by endogenous …

… for neuroprotection after brain injuries, both ischemia and traumatic brain injury, and it will
… brain injuries will pave the way for more effective therapeutic strategies after brain deficits. …

… Peroxisome proliferator-activated receptor γ (PPARγ) is a widely expressed ligand-… of
developing brain injuries such as stroke by mitigating the influence of comorbidities. If brain injury …

… We also investigated the effect of rosiglitazone treatment on post-ischemic inflammatory
responses in the brain. Our results suggest that rosiglitazone markedly decreased brain injury …

Peroxisome proliferator-activated receptor α (PPARα) has been demonstrated to reduce
inflammation in various inflammatory diseases. As traumatic brain injury … percussion of brain in …

… cell death triggered by traumatic brain injury (TBI), ferroptosis, an iron-dependent lipid
peroxidative regulatory cell death, has a critical role. Peroxisome proliferator-activated receptor-γ (…

… demonstrated that peroxisome proliferator-activated receptora (PPAR-a) agonists can protect
the brain against excessive oxidative stress and inflammation in traumatic brain injury (TBI) …

… in the inflammatory response following traumatic brain injury (TBI). Inhibiting M1 … injury
following TBI. In this study, we used a lateral fluid percussion injury device to induce axonal injury …

… In our research, the improvement of brain injury after SAH was observed in SAH+Ad-PPARβ/…
It is a known fact that neuronal apoptosis plays a major role in brain injury associated with …