Tanning is a protective response of ultraviolet (UV)-irradiated skin that decreases damage
from subsequent sun exposures by increasing the epidermal content of melanin, a brown …

T4 endonuclease V, a pyrimidine-dimer-specific DNA repair enzyme, was encapsulated in
liposomes under mild conditions. The encapsulated enzyme was active, and when applied …

Ultraviolet (UV) irradiation is a major source of environmental damage to skin. Melanin
pigmentation protects against this damage by absorbing UV photons and UV-generated free …

Ultraviolet (UV) irradiation produces two major photoproducts, cyclobutane pyrimidine
dimers (CPD) and (6–4) photoproducts. T4 endonuclease V (T4N5), which specifically …

Exposing human skin to ultraviolet radiation causes DNA damage, sunburn, immune
alterations, and eventually, skin cancer. We wished to determine whether liposomes …

Exposure of skin to ultraviolet (UV) radiation can lead to diverse biologic effects, including
inflammation, sunburn cell formation, alterations of cutaneous immune cells, and impaired …

Exposure to ultraviolet B (UVB) radiation leads to an increased generation of UVB-induced
skin damage in humans. The most important UVB-induced side effects are UVB-induced …

As sunscreens do not provide complete protection against solar/UV radiation, alternative
protective strategies are necessary to cope with the increasing incidence of skin cancer …

Tanning is commonly understood as increased melanization of the epidermis observed in
skin following UV exposure. It is further understood to represent a host response that …

Ultraviolet-B (UVB)(290–320 nm) radiation-induced cyclobutane pyrimidine dimers within
the DNA of epidermal cells are detrimental to human health by causing mutations and …