The cardinal pathology of coronavirus disease 2019 (COVID-19) is a primary infection of
pulmonary tract cells by severe acute respiratory syndrome coronavirus 2, provoking a local …

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) spread all over the world
immediately after the first patient infected with this virus was discovered in Wuhan, China, in …

Background Severe acute respiratory syndrome coronavirus 2 infection is associated with
hypercoagulability, which predisposes to venous thromboembolism (VTE). We analyzed …

Abstract Background Infection by SARS‐CoV‐2 produces significant pulmonary pathology
including endothelial damage with resultant thrombotic events. While pathologic features …

In this viewpoint, we summarize the relevance of thromboinflammation in COVID-19 and
discuss potential mechanisms of endothelial injury as a key point for the development of …

Abstract Background and Aims Patients with severe coronavirus disease 2019 (COVID‐19)
are at significant risk of thrombotic complications. However, their prothrombotic state is …

Direct endothelial injury by viruses and dysregulation of clotting mechanisms due to cytokine
storm are the major precipitating factors of mortality in COVID-19; both are attributed to a …

Background Persistent fatigue, breathlessness, and reduced exercise tolerance have been
reported following acute COVID‐19 infection. Although immuno‐thrombosis has been …

This illustrated review discusses the haemostatic changes seen in patients with severe
coronavirus disease 2019 (COVID‐19) infection and their possible causes. We discuss the …

Background: Coronavirus Disease 2019 (COVID-19)-associated coagulopathy is
characterized by a prothrombotic state not yet comprehensively studied. We investigated the …