Exposure to solar UVB radiation can lead to the formation of DNA lesions such as
cyclobutane pyrimidine dimers (CPDs). Nucleotide excision repair (NER) is critical for the …

We investigated the protective properties of diphlorethohydroxycarmalol (DPHC), a
phlorotannin, against ultraviolet B (UVB) radiation-induced cyclobutane pyrimidine dimers …

Absorption of solar ultraviolet (UV) radiation by DNA leads to the formation of the highly
mutagenic cyclobutane pyrimidine dimer (CPD). The mutagenicity of CPD is caused, in part …

Classically, the nucleotide excision repair (NER) of cyclobutane pyrimidine dimers (CPD) is
a lengthy process (t 1/2> 48 h). Using the T4 endonuclease V-modified comet assay, we …

Exposure to solar ultraviolet-B (UVB) radiation leads to the formation of cyclobutane
pyrimidine dimers (CPDs). We investigated the protective effect of apigenin against UVB …

The high incidence of skin cancers in the Caucasian population is primarily due to the
accumulation of DNA damage in epidermal cells induced by chronic ultraviolet B (UVB) …

In this study, we examined the antioxidative and the DNA protective potentials of apigenin, a
flavonoid polyphenol isolated from Lycopodium clavatum, in both in-vitro (HaCaT skin …

Ultraviolet (UV) radiation is one of the most genotoxic, universal agents present in the
environment. UVB (280-315 nm) radiation directly damages DNA, producing cyclobutane …

Solar ultraviolet (UV) radiation is a well recognized epidemiologic risk factor for melanoma
and non-melanoma skin cancers. This observation has been linked to the accumulation of …

Ultraviolet (UV) radiation from sunlight is a major etiologic factor for skin cancer, the most
prevalent cancer in the United States, as well as premature skin aging. In particular, UVB …