Extinction of conditioned fear is an active learning process requiring N-methyl-D-aspartate
receptors (NMDARs), but the timing, location, and neural mechanisms of NMDAR-mediated …

Fear extinction, which involves learning to suppress the expression of previously learned
fear, requires N-methyl-D-aspartate receptors (NMDARs) and is mediated by the amygdala …

N-methyl-D-aspartate receptors (NMDARs) contribute to synaptic plasticity underlying
learning in a variety of brain systems. Fear extinction, which involves learning to suppress …

Extinction of conditioned fear involves new learning that inhibits but does not eliminate the
original fear memory. This inhibitory learning is thought to require activation of NMDA …

We studied the roles of the basolateral amygdala (BLA) and the medial prefrontal cortex
(mPFC) in learning and relearning to inhibit context conditioned fear (freezing) in extinction …

Fear extinction does not prevent post-traumatic stress or have long-term therapeutic benefits
in fear-related disorders unless extinction memories are easily retrieved at later encounters …

Extinction of conditioned fear is thought to form a long-term memory of safety, but the neural
mechanisms are poorly understood. Consolidation of extinction learning in other paradigms …

Persistent anxiety after a psychological trauma is a hallmark of many anxiety disorders.
However, the neural circuits mediating the extinction of traumatic fear memories remain …

Anxiety disorders are commonly treated with exposure-based therapies that rely on
extinction of conditioned fear. Persistent fear and anxiety following exposure therapy could …

We have previously shown that fear extinction is accompanied by an increase of synaptic
efficacy in inputs from the ventral hippocampus (vHPC) and mediodorsal thalamus (MD) to …