The effects of prenatal exposure to ethanol on glucose utilization in specific laminae of
mature rat cerebral cortex were examined. Pregnant hooded rats were fed a liquid diet …

Cerebral cortical morphology and early motor development were evaluated in offspring from
ethanol‐exposed mothers, pairfed control mothers and ad libitum control rats. Ethanol …

One of the most severe consequences of maternal ethanol consumption is the damage to
the developing central nervous system, which is manifested by long‐term cognitive and …

The effects of maternal ethanol exposure on neurotransmission and second messenger
systems were examined in rats using histochemistry and in vitro autoradiography. Thirty …

A hallmark of fetal alcohol syndrome is microencephaly. Microencephaly can result from
either a reduction in the number of cells, in the amount of neuropil, or both. Inasmuch as …

Rats prenatally exposed to ethanol exhibit a variety of structural anomalies in the central
nervous system. One of the key features of experimental fetal alcohol syndrome is …

Prenatal exposure to ethanol causes profound disruptions in the development of the
cerebral cortex. Therefore, the effect of in utero ethanol exposure on the generation of …

The development of two forebrain fiber tracts, the corpus callosum and anterior commissure
was examined in BALB cCF and C57BL 6J mice in relation to moderate maternal …

Prenatal exposure to ethanol alters the generation of neocortical neurons; ethanol reduces
the numbers of neurons generated between gestational day (G) 12 and G19 but …

Acute maternal ethanol administration to C57B1/6J mice on gestational day 7 (GD7) results
in facial and brain abnormalities similar to those reported in human fetal alcohol syndrome …