[HTML][HTML] Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial …

S Masud, L Van der Burg, L Storm… - Frontiers in cellular …, 2019 - frontiersin.org
Intracellular pathogens such as Salmonella depend on their molecular virulence factors to
evade host defense responses like autophagy. Using a zebrafish systemic infection model …

[引用][C] Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial …

S Masud, L van der Burg, L Storm, TK Prajsnar… - Frontiers in Cellular …, 2019 - cir.nii.ac.jp
Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host
Defense Mechanism Triggered Independently From Major Bacterial Virulence Factors | CiNii …

Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial Virulence …

S Masud, L van der Burg, L Storm… - Frontiers in Cellular …, 2019 - europepmc.org
Intracellular pathogens such as Salmonella depend on their molecular virulence factors to
evade host defense responses like autophagy. Using a zebrafish systemic infection model …

[PDF][PDF] Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial …

S Masud, L van der Burg, L Storm… - Front. Cell. Infect …, 2019 - researchgate.net
Salmonella Typhimurium by an autophagy-related pathway known as Lc3-associated
phagocytosis (LAP), which is dependent on the host protein Rubicon. Here, we explore the …

Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial Virulence …

S Masud, L van der Burg, L Storm… - Frontiers in cellular …, 2019 - pubmed.ncbi.nlm.nih.gov
Intracellular pathogens such as Salmonella depend on their molecular virulence factors to
evade host defense responses like autophagy. Using a zebrafish systemic infection model …

[HTML][HTML] Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial …

S Masud, L van der Burg, L Storm… - Frontiers in Cellular …, 2019 - ncbi.nlm.nih.gov
Intracellular pathogens such as Salmonella depend on their molecular virulence factors to
evade host defense responses like autophagy. Using a zebrafish systemic infection model …

Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial Virulence …

S Masud, L van der Burg, L Storm… - Frontiers in Cellular …, 2019 - search.ebscohost.com
Intracellular pathogens such as Salmonella depend on their molecular virulence factors to
evade host defense responses like autophagy. Using a zebrafish systemic infection model …

[PDF][PDF] Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial …

S Masud, L van der Burg… - Front. Cell …, 2019 - scholarlypublications …
Salmonella Typhimurium by an autophagy-related pathway known as Lc3-associated
phagocytosis (LAP), which is dependent on the host protein Rubicon. Here, we explore the …

Rubicon-Dependent Lc3 Recruitment to Salmonella-Containing Phagosomes Is a Host Defense Mechanism Triggered Independently From Major Bacterial Virulence …

S Masud, L van der Burg, L Storm… - Frontiers in Cellular …, 2019 - europepmc.org
Intracellular pathogens such as Salmonella depend on their molecular virulence factors to
evade host defense responses like autophagy. Using a zebrafish systemic infection model …

[引用][C] Rubicon-dependent Lc3 recruitment to Salmonella-containing phagosomes is a host defense mechanism triggered independently from major bacterial …

S Masud, LLJ van der Burg… - … in Cellular and …, 2019 - scholarlypublications …
Rubicon-dependent Lc3 recruitment to Salmonella-containing phagosomes is a host defense
mechanism triggered independently from major bacterial virulence factors | Scholarly …