Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N Bye, MD Habgood, JK Callaway, N Malakooti… - Experimental …, 2007 - Elsevier
Cerebral inflammation and apoptotic cell death are two processes implicated in the
progressive tissue damage that occurs following traumatic brain injury (TBI), and strategies …

Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N Bye, MD Habgood, JK Callaway… - Experimental …, 2007 - pubmed.ncbi.nlm.nih.gov
Cerebral inflammation and apoptotic cell death are two processes implicated in the
progressive tissue damage that occurs following traumatic brain injury (TBI), and strategies …

Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N Bye, MD Habgood, JK Callaway, N Malakooti… - Experimental …, 2007 - infona.pl
Cerebral inflammation and apoptotic cell death are two processes implicated in the
progressive tissue damage that occurs following traumatic brain injury (TBI), and strategies …

[引用][C] Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N BYE - Exp Neurol, 2007 - cir.nii.ac.jp
Transient neuroprotection by minocycline following traumatic brain injury is associated with
attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration | CiNii …

[引用][C] Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N BYE, MD HABGOOD, JK CALLAWAY… - Experimental …, 2007 - pascal-francis.inist.fr
Transient neuroprotection by minocycline following traumatic brain injury is associated with
attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration CNRS …

[PDF][PDF] Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N Bye, MD Habgood, JK Callaway… - Experimental …, 2007 - academia.edu
Cerebral inflammation and apoptotic cell death are two processes implicated in the
progressive tissue damage that occurs following traumatic brain injury (TBI), and strategies …

Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N Bye, M Habgood, JK Callaway… - Experimental …, 2007 - research.monash.edu
Cerebral inflammation and apoptotic cell death are two processes implicated in the
progressive tissue damage that occurs following traumatic brain injury (TBI), and strategies …

Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N Bye, MD Habgood, JK Callaway… - Experimental …, 2006 - europepmc.org
Cerebral inflammation and apoptotic cell death are two processes implicated in the
progressive tissue damage that occurs following traumatic brain injury (TBI), and strategies …

[引用][C] Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N Bye, MD Habgood, JK Callaway, N Malakooti… - Experimental …, 2007 - cir.nii.ac.jp
Transient neuroprotection by minocycline following traumatic brain injury is associated with
attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration …

[引用][C] Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or …

N BYE, MD HABGOOD, JK CALLAWAY… - Experimental …, 2007 - Elsevier