CD38 exacerbates focal cytokine production, postischemic inflammation and brain injury after focal cerebral ischemia
C Choe, K Lardong, M Gelderblom, P Ludewig… - PLoS …, 2011 - journals.plos.org
Background Converging evidence suggests that inflammatory processes significantly
influence brain injury and clinical impairment in ischemic stroke. Although early studies
suggested a key role of lymphocytes, recent data has emphasized the orchestrating function
of innate immunity, ie, macrophages and microglia. The bifunctional receptor and
ectoenzyme CD38 synthesizes calcium-mobilizing second messengers (eg, cyclic ADP-
ribose), which have been shown to be necessary for activation and migration of myeloid …
influence brain injury and clinical impairment in ischemic stroke. Although early studies
suggested a key role of lymphocytes, recent data has emphasized the orchestrating function
of innate immunity, ie, macrophages and microglia. The bifunctional receptor and
ectoenzyme CD38 synthesizes calcium-mobilizing second messengers (eg, cyclic ADP-
ribose), which have been shown to be necessary for activation and migration of myeloid …
Correction: CD38 Exacerbates Focal Cytokine Production, Postischemic Inflammation and Brain Injury after Focal Cerebral Ischemia
C Choe, K Lardong, M Gelderblom, P Ludewig… - Plos one, 2011 - ncbi.nlm.nih.gov
Correction: CD38 Exacerbates Focal Cytokine Production, Postischemic Inflammation and
Brain Injury after Focal Cerebral Ischemia - PMC … Correction: CD38 Exacerbates Focal
Cytokine Production, Postischemic Inflammation and Brain Injury after Focal Cerebral
Ischemia … Articles from PLOS ONE are provided here courtesy of PLOS …
Brain Injury after Focal Cerebral Ischemia - PMC … Correction: CD38 Exacerbates Focal
Cytokine Production, Postischemic Inflammation and Brain Injury after Focal Cerebral
Ischemia … Articles from PLOS ONE are provided here courtesy of PLOS …
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