exacerbation of allergic respiratory diseases, including allergic rhinitis and allergic asthma.
However, DEP-induced mechanistic pathways promoting upper airway disease and their
clinical implications remain unclear. Objective We sought to investigate the mechanisms by
which DEP exposure contributes to nasal polyposis using human-derived epithelial cells
and a murine nasal polyp (NP) model. Methods Gene set enrichment and weighted gene …