The repair of oxidatively damaged DNA is integral to the maintenance of genomic stability,
and hence prevention of a wide variety of pathological conditions, such as aging, cancer
and cardiovascular disease. The ability to non-invasively assess DNA repair may provide
information regarding repair pathways, variability in repair capacity, and susceptibility to
disease. The development of assays to measure urinary DNA lesions offered this potential,
although it rapidly became clear that possible contribution from diet and cell turnover may …