The relationship between mammalian target of rapamycin complex 1 (mTORC1) signaling
and muscle protein synthesis during instances of amino acid surplus in humans is based
solely on correlational data. Therefore, the goal of this study was to use a mechanistic
approach specifically designed to determine whether increased mTORC1 activation is
requisite for the stimulation of muscle protein synthesis following L-essential amino acid
(EAA) ingestion in humans. Examination of muscle protein synthesis and signaling were …