Deinococcus radiodurans has a remarkable capacity to survive exposure to extreme levels
of radiation that cause hundreds of DNA double strand breaks (DSBs). DSB repair in this
bacterium depends on its recombinase A protein (DrRecA). DrRecA plays a pivotal role in
both extended synthesis-dependent strand annealing and slow crossover events of DSB
repair during the organism's recovery from DNA damage. The mechanisms that control
DrRecA activity during the D. radiodurans response to γ radiation exposure are unknown …