Skeletal muscle microvascular-linked improvements in glycemic control from resistance training in individuals with type 2 diabetes
RD Russell, D Hu, T Greenaway, SJ Blackwood… - Diabetes …, 2017 - Am Diabetes Assoc
OBJECTIVE Insulin increases glucose disposal in part by enhancing microvascular blood
flow (MBF) and substrate delivery to myocytes. Insulin's microvascular action is impaired
with insulin resistance and type 2 diabetes. Resistance training (RT) improves glycemic
control and insulin sensitivity, but whether this improvement is linked to augmented skeletal
muscle microvascular responses in type 2 diabetes is unknown. RESEARCH DESIGN AND
METHODS Seventeen (11 male and 6 female; 52±2 years old) sedentary patients with type …
flow (MBF) and substrate delivery to myocytes. Insulin's microvascular action is impaired
with insulin resistance and type 2 diabetes. Resistance training (RT) improves glycemic
control and insulin sensitivity, but whether this improvement is linked to augmented skeletal
muscle microvascular responses in type 2 diabetes is unknown. RESEARCH DESIGN AND
METHODS Seventeen (11 male and 6 female; 52±2 years old) sedentary patients with type …
[PDF][PDF] Skeletal Muscle Microvascular-Linked Improvements in Glycemic Control From Resistance Training in Individuals With Type 2 Diabetes
SM Richards, S Rattigan, MA Keske - 2017 - researchgate.net
OBJECTIVE Insulin increases glucose disposal in part by enhancing microvascular blood
flow (MBF) and substrate delivery to myocytes. Insulin's microvascular action is impaired
with insulin resistance and type 2 diabetes. Resistance training (RT) improves glycemic
control and insulin sensitivity, but whether this improvement is linked to augmented skeletal
muscle microvascular responses in type 2 diabetes is unknown.
flow (MBF) and substrate delivery to myocytes. Insulin's microvascular action is impaired
with insulin resistance and type 2 diabetes. Resistance training (RT) improves glycemic
control and insulin sensitivity, but whether this improvement is linked to augmented skeletal
muscle microvascular responses in type 2 diabetes is unknown.
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