the beta-lactam antibiotic ceftriaxone, represents a promising therapeutic strategy to
accelerate glutamate uptake and prevent excitotoxic damage in neurological conditions.
While excitotoxicity is indeed implicated in numerous brain diseases, it is typically restricted
to select vulnerable brain regions, particularly in early disease stages. In healthy brain
tissue, the speed of glutamate uptake is not constant and rather varies in both an activity-and …