With no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo

AS Terker, M Castañeda-Bueno… - American Journal …, 2018 - journals.physiology.org
With no lysine kinase 4 (WNK4) is essential to activate the thiazide-sensitive NaCl
cotransporter (NCC) along the distal convoluted tubule, an effect central to the phenotype of
familial hyperkalemic hypertension. Although effects on potassium and sodium channels
along the connecting and collecting tubules have also been documented, WNK4 is typically
believed to have little role in modulating sodium chloride reabsorption along the thick
ascending limb of the loop of Henle. Yet wnk4−/− mice (knockout mice lacking WNK4) do not …

With no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo

XTS Erspamer, LN Miller, JA McCormick, WH Wang… - journals.physiology.org
With no lysine kinase 4 (WNK4) is essential to activate the thiazide-sensitive NaCl
cotransporter (NCC) 29 along the distal convoluted tubule, an effect central to the phenotype
of familial hyperkalemic 30 hypertension. Although effects on potassium and sodium
channels along the connecting and collecting 31 tubules have also been documented,
WNK4 is typically believed to have little role in modulating sodium 32 chloride reabsorption
along the thick ascending limb of the loop of Henle. Yet wnk4-/-mice (knockout 33 mice …
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