Salmonella utilizes a type 3 secretion system to translocate virulence proteins (effectors) into
host cells during infection. The effectors modulate host cell machinery to drive uptake of the …

Salmonella attachment to the intestinal epithelium triggers delivery of bacterial effector
proteins into the host cytosol through a type III secretion system (T3SS), leading to …

Salmonella utilizes translocated virulence proteins (termed effectors) to promote host cell
invasion. The effector SopD contributes to invasion by promoting scission of the plasma …

Salmonella enterica serovar Typhimurium uses a type three secretion system (T3SS)
encoded on the Salmonella pathogenicity island 1 (SPI1) to invade intestinal epithelial cells …

During infection, Salmonella species inject multiple type III secretion system (T3SS) effector
proteins into host cells that mediate invasion and subsequent intracellular replication. At …

Many intracellular pathogens have evolved highly specialized mechanisms to isolate
themselves from the host cell's innate immune response while still obtaining the necessary …

The Gram-negative bacterium Salmonella enterica has developed an array of sophisticated
tools to manipulate the host cell and establish an intracellular niche, for successful …

Salmonella enterica induces membrane ruffling and genesis of macropinosomes during its
interactions with epithelial cells. This is achieved through the type three secretion system‐1 …

Salmonella is a human and animal pathogen that causes gastro-enteric diseases. The key
to Salmonella infection is its entry into intestinal epithelial cells, where the bacterium resides …

The host endolysosomal compartment is often manipulated by intracellular bacterial
pathogens. Salmonella (Salmonella enterica serovar Typhimurium) secrete numerous …