Intracellular Salmonella use a type III secretion system (TTSS) to translocate effector
proteins across the phagosome membrane and thus promote vacuole membrane tubulation …

The enteric bacterial pathogen Salmonella enterica serovar Typhimurium (S. Typhimurium),
utilizes two type III secretion systems (T3SSs) to invade host cells, survive and replicate …

Summary The Salmonella Pathogenicity Island 2 (SPI2) encodes a type III secretion system
(TTSS) shown to be critical for adaptation to the intracellular environment within both …

Salmonella enterica pathogenesis is dependent on its ability to enter and replicate inside
host cells. Replication occurs inside the Salmonella-containing vacuole (SCV), a vacuolar …

Salmonella enterica serovar Typhimurium (S. typhimurium) is a gram‐negative facultative
intracellular pathogen that can infect a broad range of mammalian hosts. Following invasion …

Outer membrane vesicles (OMVs) are spherical membranous structures released by Gram-
negative bacteria. Several bacterial pathogens utilize OMVs as vehicles for the delivery of …

After Salmonella is phagocytosed, it resides in an acidic vacuole. Its cytoplasm acidifies to
pH 5.6; acidification activates pathogenicity island 2 (SPI-2). SPI-2 encodes a type three …

Salmonella pathogenesis is dependent on its ability to invade and replicate within host cells.
Following invasion the bacteria remain within a modified phagosome known as the …

The Salmonella invasion-associated type III secretion system (T3SS1) is an essential
virulence factor required for entry into nonphagocytic cells and consequent uptake into a …

Salmonella enterica serovar Typhimurium utilizes a type III secretion system (TTSS)
encoded on Salmonella pathogenicity island-2 (SPI2) to promote intracellular replication …