Rationale: A rapid and massive influx of inflammatory cells occurs into ischemic area after
myocardial infarction (MI), resulting in local release of cytokines and growth factors. Yet, the …

Extracellular vesicles (EVs) curb important biological functions. We previously disclosed that
ischemia‐reperfusion (IR) induces increased release of EVs (IR‐EVs) in the heart. However …

After myocardial infarction (MI), a strong inflammatory response takes place in the heart to
remove the dead tissue resulting from ischemic injury. A growing body of evidence suggests …

Aims Increased shedding of extracellular vesicles (EVs)—small, lipid bilayer-delimited
particles with a role in paracrine signalling—has been associated with human pathologies …

Myocardial infarction (MI) triggers a potent inflammatory response via the release of
circulatory mediators, including extracellular vesicles (EVs) by damaged cardiac cells …

Extracellular vesicles (EVs)—particularly exosomes and microvesicles (MVs)—are attracting
considerable interest in the cardiovascular field as the wide range of their functions is …

Background: After myocardial infarction, necrotic cardiomyocytes release damage-
associated proteins that stimulate innate immune pathways and macrophage tissue …

Extracellular vesicles are heterogeneous structures surrounded by cell membranes and
carry complex contents including nucleotides, proteins, and lipids. These proteins include …

Extracellular vesicles (EVs) serve an important function as mediators of intercellular
communication. Exercise is protective for the heart, although the signaling mechanisms that …

Plasma circulating extracellular vesicles (EVs) have been utilized as a potential therapeutic
strategy to treat ischemic disease through intramyocardial injection (efficient but invasive) or …