The biological relevance of extracellular vesicles (EV) released in an ischemia/reperfusion
setting is still unclear. We hypothesized that the inflammatory microenvironment prevents …

Rationale: A rapid and massive influx of inflammatory cells occurs into ischemic area after
myocardial infarction (MI), resulting in local release of cytokines and growth factors. Yet, the …

Extracellular vesicles (EVs) serve an important function as mediators of intercellular
communication. Exercise is protective for the heart, although the signaling mechanisms that …

During myocardial ischemia, timely reperfusion is critical to limit infarct area and the overall
loss of cardiac contractile function. However, reperfusion further exacerbates the damage of …

Extracellular vesicles (EVs) curb important biological functions. We previously disclosed that
ischemia‐reperfusion (IR) induces increased release of EVs (IR‐EVs) in the heart. However …

Abstract Background Extracellular vesicles (EVs) derived from various cell sources exert
cardioprotective effects during cardiac ischemic injury. Our previous study confirmed that …

Myocardial ischaemia is associated with an exacerbated inflammatory response, as well as
with a deregulation of intercellular communication systems. Macrophages have been …

Extracellular vesicles (EVs) derived from various stem cell sources induce cardioprotective
effects during ischemia-reperfusion injury (IRI). These have been attributed mainly to the …

Abstract Rationale Extracellular vesicles (EVs) derived exogenously from pluripotent stem
cells or endogenously from healthy human serum exert cardioprotective effects after injury …

Introduction Wound healing after myocardial infarction (MI) is mediated by different cell
types, secreted proteins, components of the extracellular matrix (ECM) and, as increasing …