BACKGROUND: The immune system's role in ST-segment–elevated myocardial infarction
(STEMI) remains poorly characterized but is an important driver of recurrent cardiovascular …

Myocardial infarction (MI) is strongly associated with the temporal regulation of cardiac
immunity. However, a variety of current clinical trials have failed because of the lack of post …

Background Myocardial infarction (MI) is characterized by the emergence of dead or dying
cardiomyocytes and excessive immune cell infiltration after coronary vessel occlusion …

Acute myocardial infarction (MI) causes sterile inflammation, which is characterized by
recruitment and activation of innate and adaptive immune system cells. Here we delineate …

Background Acute myocardial infarction (AMI) can occur in patients with atherosclerotic
disease, with or without plaque rupture. Previous studies have indicated a set of immune …

Introduction Chronic immunopathology contributes to the development of heart failure after a
myocardial infarction. Both T and B cells of the adaptive immune system are present in the …

An increasing body of evidence has implicated the innate immune system in the causation of
acute ST-segment elevation myocardial infarction (STEMI). Innate lymphoid cells (ILCs) are …

Introduction Myocardial infarction (MI) is a deadly medical condition leading to irreversible
damage to the inflicted cardiac tissue. Elevated inflammatory response marks the severity of …

Despite recent scientific and technological advances, myocardial infarction (MI) still
represents a major global health problem, leading to high morbidity and mortality worldwide …

Sepsis is a syndrome of life-threatening organ dysfunc-tion caused by a dysregulated host
response to infection. Leukocyte gene expression suggests that although most septic …