[HTML][HTML] m 6A methylation in cellular senescence of age-associated diseases: m 6A methylation in cellular senescence of age-associated diseases
P Gao, F Yao, J Pang, K Yin, X Zhu - Acta Biochimica et Biophysica …, 2023 - ncbi.nlm.nih.gov
Cellular senescence is a state of irreversible cellular growth arrest that occurs in response to
various stresses. In addition to exiting the cell cycle, senescent cells undergo many …
various stresses. In addition to exiting the cell cycle, senescent cells undergo many …
[HTML][HTML] RNA epigenetics in pulmonary diseases: Insights into methylation modification of lncRNAs in lung cancer
J Jiang, M Duan, Z Wang, Y Lai, C Zhang… - Biomedicine & …, 2024 - Elsevier
Long non-coding RNAs (lncRNAs) are pivotal controllers of gene expression through
epigenetic mechanisms, Methylation, a prominent area of study in epigenetics, significantly …
epigenetic mechanisms, Methylation, a prominent area of study in epigenetics, significantly …
Changes in M6A methylation: a key factor in the vicious cycle of flora-gut aging
M Ding, J Yan, Y Chen, J Liu, G Chao… - Ageing Research Reviews, 2024 - Elsevier
The aging process significantly impacts the gastrointestinal tract and various bodily systems,
exacerbating age-related diseases. Research suggests a correlation between an imbalance …
exacerbating age-related diseases. Research suggests a correlation between an imbalance …
Negative Regulation of LINC01013 by METTL3 and YTHDF2 Enhances the Osteogenic Differentiation of Senescent Pre‐Osteoblast Cells Induced by Hydrogen …
J Song, Y Wang, Z Zhu, W Wang, H Yang… - Advanced …, 2024 - Wiley Online Library
Senescent pre‐osteoblasts have a reduced ability to differentiate, which leads to a reduction
in bone formation. It is critical to identify the keys that regulate the differentiation fate of …
in bone formation. It is critical to identify the keys that regulate the differentiation fate of …