[HTML][HTML] APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - cell.com
Summary Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic
Alzheimer's disease (sAD). Inflammation is increasingly considered as critical in sAD …

APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation.

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - europepmc.org
Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic Alzheimer's
disease (sAD). Inflammation is increasingly considered as critical in sAD initiation and …

APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - inria.hal.science
Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic Alzheimer's
disease (sAD). Inflammation is increasingly considered as critical in sAD initiation and …

APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell …, 2022 - hal.insa-toulouse.fr
Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic Alzheimer's
disease (sAD). Inflammation is increasingly considered as critical in sAD initiation and …

[引用][C] APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - cir.nii.ac.jp
APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation
| CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ[サイニィ] 詳細へ移動 検索フォームへ …

[PDF][PDF] APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-kB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell …, 2022 - scholar.archive.org
SUMMARY Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic
Alzheimer's disease (sAD). Inflammation is increasingly considered as critical in sAD …

APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - cnrs.hal.science
Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic Alzheimer's
disease (sAD). Inflammation is increasingly considered as critical in sAD initiation and …

APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - hal.science
Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic Alzheimer's
disease (sAD). Inflammation is increasingly considered as critical in sAD initiation and …

APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - hal.univ-smb.fr
Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic Alzheimer's
disease (sAD). Inflammation is increasingly considered as critical in sAD initiation and …

[HTML][HTML] APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activation

L Arnaud, P Benech, L Greetham, D Stephan… - Cell Reports, 2022 - cell.com
Summary Apolipoprotein E4 (APOEε4) is the major allelic risk factor for late-onset sporadic
Alzheimer's disease (sAD). Inflammation is increasingly considered as critical in sAD …