Amyloid β-peptide (Aβ) aggregation is a critical step in the pathogenesis of Alzheimer's
disease (AD). Inhibition of Aβ production, dissolution of existing aggregates and clearance …

The fibrillization and abnormal aggregation of β-amyloid (A β) peptides are commonly
recognized risk factors for Alzheimer's disease (AD) brain, and require an effective strategy …

The self‐assembly of amyloidogenic peptides into β‐sheet‐rich aggregates is a general
feature of many neurodegenerative diseases, including Alzheimer's disease, which signifies …

Despite the accumulating evidence linking the development of Alzheimer's disease (AD) to
the aggregation of Aβ peptides and the emergence of Aβ oligomers, the FDA has approved …

Amyloid protein misfolds, abnormally aggregates and accumulates into amyloid deposits
which endanger tissue functions and are closely related to the pathogenesis of many …

Amyloid aggregation and fungal infection, especially amyloid beta (Aβ) peptide and
Candida albicans are considered as two of the crucial pathogenic agents in Alzheimer's …

The accumulation and deposits of amyloid beta (Aβ) peptide are an important pathological
hallmark of Alzheimer's disease (AD). The development of multifunctional agents that can …

The abnormal aggregation of β-amyloid protein (Aβ) is a major contributor to Alzheimer's
disease (AD). Cu 2+ homeostasis imbalance can lead to the aggregation of Aβ, resulting in …

The design of high-efficiency scavengers targeting β-amyloid protein (Aβ) plaques in the
progress of Alzheimer's disease (AD) has been recognized as an effective way to prevent …

The accumulation of amyloid-β (Aβ) peptides is a major hallmark of Alzheimer's disease
(AD) and plays a crucial role in its pathogenesis. Particularly, the structured oligomeric …