Abstract Fine particulate matter (PM2. 5) concentrations have decreased in the past decade.
The adverse effects of acute PM2. 5 exposure on respiratory diseases have been well …

Background Long-term exposure to fine particulate matter (PM2. 5) increases susceptibility
to chronic respiratory diseases, including inflammation and interstitial fibrosis. However, the …

Abstract Fine particulate matter (PM2. 5) is the primary air pollutant that is able to induce
airway injury. Compelling evidence has shown the involvement of IL‐17A in lung injury …

Exposure to fine atmospheric particulate matter (PM) is known to induce lung inflammation
and injury; however, the way in which sophisticated endogenous lung repair and …

Inflammatory responses are considered to be the critical mechanism underlying particulate
matter (PM)-induced development and exacerbation of chronic respiratory diseases. MiR …

The impacts of ambient fine particulate matter (PM 2.5) on public health are a worldwide
concern. Epidemiological evidence has shown that PM 2.5-triggered inflammatory cascades …

The relationship between fine particulate matter (PM2. 5) and chronic airway inflammatory
diseases, such as chronic obstructive pulmonary disease and asthma, have garnered public …

Particulate matter (PM) is associated with the incidence, exacerbation, and mortality of
variable respiratory diseases. However, the molecular mechanisms of PM 10-mediated …

Exposure to atmospheric particulate matter (PM) has been related to the increasing
incidence and mortality of pulmonary diseases, where microRNAs (miRNAs) play significant …

Abstract Exposure to particulate matter 2.5 (PM2. 5) potentially triggers airway inflammation
by activating nuclear factor-κB (NF-κB). Sirtuin 2 (SIRT2) is a key modulator in inflammation …