Cell-mediated immunity in experimental Trypanosoma cruzi infection
GA DosReis - Parasitology today, 1997 - cell.com
Infection of humans with the protozoan Trypanosoma cruzi leads to Chagas disease, or
American trypanosomiasis, a disease that affects nearly 20 million people, and constitutes …
American trypanosomiasis, a disease that affects nearly 20 million people, and constitutes …
Trypanosoma brucei brucei: a long-term model of human African trypanosomiasis in mice, meningo-encephalitis, astrocytosis, and neurological disorders
M Keita, B Bouteille, B Enanga, JM Vallat… - Experimental …, 1997 - Elsevier
The search for a chronic experimental model for human African trypanosomiasis (HAT) in
animals with cerebral lesions and neurological disorders has been difficult. Models with …
animals with cerebral lesions and neurological disorders has been difficult. Models with …
The gene for a T lymphocyte triggering factor from African trypanosomes
T Vaidya, M Bakhiet, KL Hill, T Olsson… - The Journal of …, 1997 - rupress.org
An early and essential event in the protective immune response against most viruses and
protozoa is the production of interferon-γ (IFN-γ). In contrast, during infection with African …
protozoa is the production of interferon-γ (IFN-γ). In contrast, during infection with African …
Trypanosoma cruzi induces strong IL-12 and IL-18 gene expression in vivo: correlation with interferon-gamma (IFN-γ) production
C Meyer Zum Büschenfelde, S Cramer… - Clinical & …, 1997 - academic.oup.com
SUMMARY IFN-γ, produced after infection with Trypanosoma cruzi, has been shown to be
crucial in the determination of resistance or susceptibility. We have performed a detailed …
crucial in the determination of resistance or susceptibility. We have performed a detailed …
The identification and molecular characterization of Trypanosoma cruzi amastigote surface protein-1, a member of the trans-sialidase gene super-family
MAM Santos, N Garg, RL Tarleton - Molecular and biochemical …, 1997 - Elsevier
An accumulating body of evidence suggests that T. cruzi-infected host cells are recognized
and destroyed by class I major histocompatibility complex (MHC) restricted CD8+ T-cells …
and destroyed by class I major histocompatibility complex (MHC) restricted CD8+ T-cells …
Trypanosoma cruzi infection does not impair major histocompatibility complex class I presentation of antigen to cytotoxic T lymphocytes
FS Buckner, BT Wipke… - European journal of …, 1997 - Wiley Online Library
Abstract Trypanosoma cruzi (T. cruzi), the etiological agent of Chagas' disease, lives free
within the cytoplasm of infected host cells. This intracellular niche suggests that parasite …
within the cytoplasm of infected host cells. This intracellular niche suggests that parasite …
Infection with Trypanosoma cruzi selectively upregulates B7-2 molecules on macrophages and enhances their costimulatory activity
S Frosch, D Küntzlin, B Fleischer - Infection and immunity, 1997 - Am Soc Microbiol
T-cell-mediated immune responses are essential for protection against infection with the
protozoan Trypanosoma cruzi. In this study, we investigated the influence of infection of …
protozoan Trypanosoma cruzi. In this study, we investigated the influence of infection of …
Trypanosoma cruzi: resistance to the pore forming protein of cytotoxic lymphocytes—perforin
The pore-forming protein perforin is one of the main effector molecules which cytotoxic
lymphocytes utilize to kill their targets bothin vivoandin vitro. Natural killer cells and cytotoxic …
lymphocytes utilize to kill their targets bothin vivoandin vitro. Natural killer cells and cytotoxic …
Identification and characterization of a putative host cell receptor for Trypanosoma cruzi trypomastigote involved in trypanosome binding and entry
A Valentine - 1997 - search.proquest.com
Trypanosome entry is an unique and remarkable process in membrane biology that is as yet
not understood. Molecules involved in the process by which Trypanosoma cruzi invade …
not understood. Molecules involved in the process by which Trypanosoma cruzi invade …