Uptake of β-N-methylamino-L-alanine (BMAA) into glutamate-specific synaptic vesicles: Exploring the validity of the excitotoxicity mechanism of BMAA
R van Onselen, TG Downing - Neuroscience Letters, 2024 - Elsevier
The first mechanism of toxicity proposed for the cyanobacterial neurotoxin β-N-methylamino-
L-alanine (BMAA) was excitotoxicity, and this was supported by numerous in vitro studies in …
L-alanine (BMAA) was excitotoxicity, and this was supported by numerous in vitro studies in …
[HTML][HTML] Protection against β-N-methylamino-l-alanineꟷinduced vesicular monoamine transporter 2 inhibition by hydroxyl-containing proteinogenic amino acids
R van Onselen, C Kennedy, TG Downing - Environmental Toxicology and …, 2024 - Elsevier
Abstract β-N-methylamino-l-alanine (BMAA) has been shown to inhibit vesicular monoamine
transporter 2 (VMAT2), thereby preventing the uptake of monoaminergic neurotransmitters …
transporter 2 (VMAT2), thereby preventing the uptake of monoaminergic neurotransmitters …
β-N-Methylamino-L-Alanine (BMAA) Modulates the Sympathetic Regulation and Homeostasis of Polyamines
The neurotoxin β-N-methylamino-L-alanine (BMAA) is a non-proteinogenic amino acid
produced by cyanobacteria. Non-neuronal toxicity of BMAA is poorly studied with a reported …
produced by cyanobacteria. Non-neuronal toxicity of BMAA is poorly studied with a reported …