Exploring extramedullary hematopoiesis: unraveling the hematopoietic microenvironments

G Rivera-Torruco, MO Muench… - Frontiers in Hematology, 2024 - frontiersin.org
Hematopoiesis is a process by which all blood cells are formed. The mechanisms controlling
it have been studied for decades. Surprisingly, while hematopoietic stem cells are among …

Iron Overload in a Patient with Non-Transfusion-Dependent Hemoglobin H Disease and Borderline Serum Ferritin: Can We Rely on Serum Ferritin for Monitoring in …

M Ali, MA Yassin, M Aldeeb - Case Reports in Oncology, 2020 - karger.com
Secondary iron overload is a common complication in the context of hematological
diseases, as iron accumulates due to different mechanisms including chronic transfusion …

Extramedullary hematopoiesis in β-thalassemia major patient: a case report and review of the literature

B Keikhaei, D Purrahman, B Marashi, M Moezi… - Journal of …, 2022 - Springer
Extramedullary hematopoiesis (EMH), as a compensatory phenomenon, refers to the blood
cell formation outside of the bone marrow that occurs once the cells in the circulatory system …

Luspatercept's use in a patient with transfusion‐dependent beta‐thalassemia and intrathoracic extramedullary hematopoiesis (EMH)

MN Seijari, A Alshurafa, MA Yassin - Clinical Case Reports, 2024 - Wiley Online Library
Key Clinical Message This case report and literature review examine the use of a relatively
novel agent in a transfusion‐dependent beta‐thalassemia patient with extramedullary …

Low back pain in beta thalassemia major revealing sacral extramedullay hematopoeisis: a case report

RW Ahmad, LA Okar, A Elhiday, H Almasri… - Clinical Case …, 2021 - Wiley Online Library
Extramedullary hematopoiesis (EMH) is a well‐known complication of beta thalassemia
major and frequently occurs in typical sites such as liver or spleen. However, when …

Addison's disease in a lady with hemoglobin H disease

NG Rashid - Iraqi Journal of Hematology, 2022 - journals.lww.com
Hemoglobin H (HbH) disease is alpha (α)-thalassemia characterized by the inactivation of
three of four α-globin genes due to deletions with or without nondeletional α-thalassemia …