Varicella-zoster virus (VZV) causes clinically significant illness during acute and recurrent
infection accompanied by robust innate and acquired immune responses. Innate immune …

Primary varicella-zoster virus (VZV) infection causes varicella (chickenpox) and the
establishment of a lifelong latent infection in ganglionic neurons. VZV reactivates in about …

Satellite glial cells (SGCs) are homeostatic cells enveloping the somata of peripheral
sensory and autonomic neurons. A wide variety of neuronal stressors trigger activation of …

Herpesviruses hijack the MHC class I (MHC I) and class II (MHC II) antigen-presentation
pathways to manipulate immune recognition by T cells. First, we illustrate herpes simplex …

Varicella zoster virus (VZV) is a neurotropic alphaherpesvirus and the causative agent of
varicella (chickenpox) in humans. Following primary infection, VZV establishes latency in the …

Primary infection with varicella zoster virus (VZV), an exclusively human neurotrophic
alphaherpsesvirus, results in varicella, known more commonly as chickenpox. Like other …

Introduction Varicella zoster virus (VZV) causes varicella and can reactivate as herpes
zoster, and both diseases present a significant burden worldwide. However, the …

Satellite glial cells (SGCs) are the main type of glial cells in sensory ganglia. Animal studies
have shown that these cells play essential roles in both normal and disease states. In a …

Although the establishment, maintenance and reactivation from alphaherpesvirus latency is
far from fully understood, some things are now manifestly clear: Alphaherpesvirus latency …

Primary infection with varicella-zoster virus (VZV), a neurotropic alphaherpesvirus, results in
varicella. VZV establishes latency in the sensory ganglia and can reactivate later in life to …