Redox signaling regulates physiological self-renewal, proliferation, migration and
differentiation in gastrointestinal epithelium by modulating Wnt/β-catenin and Notch …

Crohn's disease (CD) and ulcerative colitis (UC), known as inflammatory bowel disease
(IBD), are fairly common chronic inflammatory conditions of the gastrointestinal tract …

To propose a new pathogenesis called Radical Induction to explain the genesis and
progression of ulcerative colitis (UC). UC is an inflammatory bowel disease. Colonic …

Background and aims: Leukocyte infiltration, up-regulation of proinflammatory cytokines and
severe oxidative stress caused by increased amounts of reactive oxygen species are …

The relationship between motility and inflammatory gastrointestinal disorders is at the same
time complex and intriguing since these conditions might share some genetic …

Gastroesophageal reflux disease complicated by Barrett esophagus (BE) is a major risk
factor for esophageal adenocarcinoma (EA). The mechanisms whereby acid reflux may …

Oxidative stress is involved in the initiation and propagation of chronic intestinal pathologies.
Bioactive peptides such as egg yolk-derived phosvitin phosphopeptides (PPP3) have been …

Tachykinins (TKs), substance P (SP), neurokinin A (NKA) and B (NKB) are important peptide
modulators of intestinal motility in animal species studied so far, including humans …

The role of oxidants and reactive nitrogen species in irritable bowel syndrome: A potential
etiological explanation - PMC Back to Top Skip to main content NIH NLM Logo Access keys …

In this comprehensive evidence-based analysis of ulcerative colitis (UC), a causal role is
identified for colonic epithelial hydrogen peroxide (H 2 O 2) in both the pathogenesis and …