This article reviews current knowledge on human and porcine cysticercosis caused by
Taenia solium. It highlights the conditions favorable for its prevalence and transmission, as …

Recent experimental evidence suggests that parasites can not only evade immune
responses actively but also exploit the hormonal microenvironment within the host to favor …

Sex biases in parasitism may be expected in mammals because estrogens stimulate
immunity, whereas androgens depress immunity. Parasites should, therefore, become more …

In early stages of experimental murine cysticercosis caused by Taenia crassiceps, there is a
clear but transient Th1-type immune response (characterized by high levels of interleukin [IL] …

Female mice are more susceptible to Taenia crassiceps (TC) infection than males. However,
after a month parasite load increases massively in both genders reaching thousands of …

A review of current literature on mammalian hosts' sexual dimorphism (SD) in parasitic
infections revealed that (1) it is a scarcely and superficially studied biological phenomenon …

Human infection with the protozoan parasite Trypanosoma cruzi leads to Chagas disease,
which affects∼ 17 million people in Latin America. A significant percentage of the infected …

Infection heterogeneity is one of the most fundamental patterns in disease ecology, yet
surprisingly few studies have experimentally explored its underlying drivers. Here, we used …

The in vitro exposure of Taenia crassiceps cysticerci to 17-β estradiol (E2) and progesterone
(P4) stimulated their reproduction and infectivity. Testosterone (T4) and dihydrotestosterone …

Prominent estrogenization and deandrogenization ensue in male mice as a consequence of
experimental intraperitoneal infection with Taenia crassiceps cysticerci. The impact of these …