Introduction Hepatic lipid accumulation and mitochondrial dysfunction are hallmarks of
metabolic associated fatty liver disease (MAFLD), yet molecular parameters underlying …

Accumulating evidence reveal that maternal smoking or perinatal nicotine replacement
therapy impairs hippocampal neurogenesis, neural development, and cognitive behaviors in …

Polycyclic aromatic hydrocarbons (PAHs), as a kind of endocrine disruptors, can enter the
fetus body cross the placental barrier from prenatal PAHs exposure to cause adverse birth …

Prenatal and early-life exposure to cigarette smoke (CS) has repeatedly been shown to
induce stable, long-term changes in DNA methylation (DNAm) in offspring. It has been …

Background Childhood exposure to polycyclic aromatic hydrocarbons (PAHs) or lead (Pb) is
associated with epigenetic modifications. However, the effects of their co-exposures on IGF1 …

Highlights•The e-waste exposed pregnant women have a greater chronic daily intake of PM
2.5.•Maternal PM 2.5 exposure is associated with neonatal BAI1 methylation at 2 …

Objective Although population-based studies of intrauterine growth restriction (IUGR)
demonstrated a series of postnatal complications, several studies identified that IUGR could …

Carbon black nanoparticles (CBNPs) are one of the most frequently used nanoparticles.
Exposure to CBNPs during pregnancy (PrE to CBNPs) can directly induce inflammation …

Abstracts Introduction and objectives Gallbladder disease is a common disease with high
prevalence. Majority of gallbladder disease is due to gallstone. Though genetics are …

DNA methylation patterns derived from fetal vulnerability to maternal smoking relate to
future child outcomes Page 1 DNA methylation patterns derived from fetal vulnerability to …