The cytokine network in chronic obstructive pulmonary disease
PJ Barnes - American journal of respiratory cell and molecular …, 2009 - atsjournals.org
Multiple cytokines play a role in the orchestration of inflammation in inflammatory airway
diseases, such as chronic obstructive pulmonary disease, through the recruitment, activation …
diseases, such as chronic obstructive pulmonary disease, through the recruitment, activation …
Current concepts on oxidative/carbonyl stress, inflammation and epigenetics in pathogenesis of chronic obstructive pulmonary disease
H Yao, I Rahman - Toxicology and applied pharmacology, 2011 - Elsevier
Chronic obstructive pulmonary disease (COPD) is a global health problem. The current
therapies for COPD are poorly effective and the mainstays of pharmacotherapy are …
therapies for COPD are poorly effective and the mainstays of pharmacotherapy are …
Interleukin 32, inflammation and cancer
JT Hong, DJ Son, CK Lee, DY Yoon, DH Lee… - Pharmacology & …, 2017 - Elsevier
Abstract Interleukin-32 (IL-32) is a novel cytokine involved in inflammation and cancer
development. IL-32 gene consists of eight small exons, and IL-32 mRNA has nine …
development. IL-32 gene consists of eight small exons, and IL-32 mRNA has nine …
Epithelial decision makers: in search of the'epimmunome'
Frequent microbial and nonmicrobial challenges to epithelial cells trigger discrete pathways,
promoting molecular changes such as the secretion of specific cytokines and chemokines …
promoting molecular changes such as the secretion of specific cytokines and chemokines …
Potent phagocytic activity with impaired antigen presentation identifying lipopolysaccharide-tolerant human monocytes: demonstration in isolated monocytes from …
C del Fresno, F García-Rio, V Gómez-Pina… - The Journal of …, 2009 - journals.aai.org
Monocyte exposure to LPS induces a transient state in which these cells are refractory to
further endotoxin stimulation. This phenomenon, termed endotoxin tolerance (ET), is …
further endotoxin stimulation. This phenomenon, termed endotoxin tolerance (ET), is …
Histone deacetylase inhibitors for treating a spectrum of diseases not related to cancer
CA Dinarello, G Fossati, P Mascagni - Molecular medicine, 2011 - Springer
Abstract This issue of Molecular Medicine contains 14 original research reports and state-of-
the-art reviews on histone deacetylase inhibitors (HDACi's), which are being studied in …
the-art reviews on histone deacetylase inhibitors (HDACi's), which are being studied in …
Subtyping of polyposis nasi: phenotypes, endotypes and comorbidities
M Koennecke, L Klimek, J Mullol, P Gevaert… - Allergo journal …, 2018 - Springer
Background Chronic rhinosinusitis (CRS) is a heterogeneous, multifactorial inflammatory
disease of the nasal and paranasal mucosa. It has not been possible to date to develop an …
disease of the nasal and paranasal mucosa. It has not been possible to date to develop an …
IL-32 is expressed by human primary keratinocytes and modulates keratinocyte apoptosis in atopic dermatitis
N Meyer, M Zimmermann, S Bürgler, C Bassin… - Journal of Allergy and …, 2010 - Elsevier
BACKGROUND: Keratinocyte (KC) apoptosis is an important mechanism of eczema and
spongiosis in patients with atopic dermatitis (AD) and is mediated by IFN-γ, which is …
spongiosis in patients with atopic dermatitis (AD) and is mediated by IFN-γ, which is …
Identification of cells expressing IL-17A and IL-17F in the lungs of patients with COPD
A Eustace, LJC Smyth, L Mitchell, K Williamson… - Chest, 2011 - Elsevier
Background Lymphocytes secrete IL-17A and IL-17F, which enhance innate immune
responses. IL-17 expression has not been studied in COPD small airways. The aim of this …
responses. IL-17 expression has not been studied in COPD small airways. The aim of this …
IL-32: a host proinflammatory factor against influenza viral replication is upregulated by aberrant epigenetic modifications during influenza A virus infection
Our previous studies with clinical data analysis have shown that the proinflammatory factor
IL-32 is activated in response to influenza virus infection. However, little is known about how …
IL-32 is activated in response to influenza virus infection. However, little is known about how …